Thursday, August 4, 2016

UPCOMING AGENT ORANGE TOWN HALL MEETINGS












August 6, 2016
Appleton, Wisconsin
Contact: Michael Demske

August 25, 2016
Long Beach, California
Contact: Max Stewart
  
August 27, 2016
St. Louis, Missouri
Contact: Bill Kiefer

September 18, 2016
Kenilworth, New Jersey
Michael Eckstein, Chair

October 1, 2016
Tulsa, Oklahoma
Contact: Nate Washington
Lew Broughton


Wednesday, August 3, 2016

Agent Orange Links

courtesy of our old friend, George Claxton

Agent Orange Links August 2, 2016

Divergent Effects of Dioxin- or Non-Dioxin-Like Polychlorinated Biphenyls on the Apoptosis of Primary Cell Culture from the Mouse Pituitary Gland
Reproductive toxicology of 2, 4 dinitrophenol in boar sperm
Dioxins reformation and destruction in secondary copper smelting fly ash under ball milling.
A study on phthalate metabolites, bisphenol A and nonylphenol in the urine of Chinese women with unexplained recurrent spontaneous abortion.
Parabens and Human Epidermal Growth Factor Receptor Ligand Cross-Talk in Breast Cancer Cells
State of the art and advances in the impact assessment of dioxins and dioxin-like compounds.
Dioxin Key Facts
Effect of colestimide on the concentrations of polychlorinated dibenzo-p-dioxins, polychlorinated dizenzofurans, and polychlorinated biphenyls in blood of Yusho patients
Toxicology Litigation Support
Dioxin emissions from a municipal solid waste incinerator and risk of invasive breast cancer: a population-based case-control study with GIS-derived exposure
Polychlorinated biphenyl exposure alters the expression profile of microRNAs associated with vascular diseases.
Arsenic-induced dose-dependent modulation of the NF-κB/IL-6 axis in thymocytes triggers differential immune responses.
Perfluoroalkyl Acids in Maternal Serum and Indices of Fetal Growth: The Aarhus Birth Cohort
Estimation of Total and Inorganic Arsenic Intake from the Diet in Korean Adults.
Mechanism-specific injury biomarkers predict nephrotoxicity early following glyphosate surfactant herbicide (GPSH) poisoning.
Diesel exhaust: current knowledge of adverse effects and underlying cellular mechanisms
Alterations in the reproductive patterns of female mice exposed to xenobiotics.
Neonatal exposure to a glyphosate based herbicide alters the development of the rat uterus
Infant Infections and Respiratory Symptoms in Relation to in Utero Arsenic Exposure in a U.S. Cohort
Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis
Impeded Immunity? Reduced Tuberculosis Vaccine Response with Exposure to Environmental Chemicals
Serum PCB Concentrations and Cochlear Function in 12-Year-Old Children
Dioxin-like activities in serum across European and Inuit populations
Beyond toxicity: aryl hydrocarbon receptor-mediated functions in the immune system
An autophagic process is activated in HepG2 cells to mediate BDE-100-induced toxicity.
Pollutants in pet dogs: a model for environmental links to breast cancer.
Major congenital malformations and residential proximity to a regional industrial park including a national toxic waste site: An ecological study
Prenatal ambient air pollution exposure and the risk of stillbirth: systematic review and meta-analysis of the empirical evidence
Metabolically inert perfluorinated fatty acids directly activate uncoupling protein 1 in brown-fat mitochondria
Cellular and molecular biology of aryl hydrocarbon (Ah) receptor-mediated gene expression.
Evaluation of relative effect potencies (REPs) for dioxin-like compounds to derive systemic or human-specific TEFs to improve human risk assessment.
Dioxins and Dioxin-Like Compounds in Food and Feed
Diesel exhaust: current knowledge of adverse effects and underlying cellular mechanisms

No Relief for Some Vets Exposed to Agent Orange

(CN) — The D.C. Circuit upheld the dismissal of "blue water" Vietnam veterans' claim that the Department of Veterans Affairs wrongly refused to compensate them for injuries caused by Agent Orange exposure.
The U.S. military used Agent Orange as a defoliant to clear forested areas throughout the Vietnam War, and thousands of soldiers were exposed to the herbicide mixture.
A list of potential health consequences posted on the VA's website notes that exposure can lead to increased rates of acute leukemia, Hodgkin's lymphoma and non-Hodgkin's lymphoma, throat cancer, prostate cancer, lung cancer, colon cancer, Ischemic heart disease, soft tissue sarcoma and liver cancer.
Exposure to Agent Orange was so prevalent that Congress eventually recognized the long-term health effects of the chemical and passed the Agent Orange Act of 1991, which creates a presumption that veterans who served between 1962 and 1975 were exposed.
If these veterans develop diseases linked to Agent Orange, they can receive disability compensation without proving they were exposed to the herbicide.
However, the VA does not offer such benefits to veterans who served on ships offshore, never set foot on Vietnamese soil, or served on ships that entered inland waterways.
Instead, these "blue-water" veterans must prove exposure on a case-by-case basis. The Blue Water Navy Vietnam Veterans Association unsuccessfully challenged the distinction in a 2015 lawsuit.
The veterans noted that the presence of Agent Orange in the waters off the coast of Vietnam was unmistakable.
"Whenever ships anchored, the anchoring evolution would disturb the shallow seabed and churn up the bottom," they claimed. "Weighing anchor actually pulled up a small portion of the bottom. The cavitation of military ships moving along the coast line, especially within the 10 fathom curve, at high speeds, further impinged on the sea bottom. This caused the Agent Orange to constantly rise to the surface."